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NUTRITION IN SURGICAL PATIENTS
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ENTERAL NUTRITION
Malnutrition in surgical patients is driven by the STRESS response and affects upto 60% of surgical patients on admission.
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SUPPLEMENTATION of oral intake -
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N.I.C.E. describes malnutrition as -
Where possible, feeding by enteral route should be maintained.
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Body mass index (B.M.I) <18 Kg/m2
Carbohydrate/ protein supplements should be used as ADJUNCT to oral intake
Unintentional weight loss > 10% within last 3-6 months
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B.M.I. <20Kg/m2 + unintentional weight loss >5% in the last 3-6 months
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Naso-Gastric feeding -
Stress response to surgery/critical illness is a myriad of metabolic and hormonal changes that create a CATABOLIC
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Indications -
state. Release of catecholamines causes Tachycardia + vasoconstriction. R.A.A.S activation causes sodium and water
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If oral intake is deemed insufficient
retention. Cortisol causes hyPERglycemia, proteolysis and lipolysis. GH promotes protein synthesis and along with cortisol
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Oral intake is deemed unsafe - Swallow problems, altered consciousness, etc
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causes insulin resistance.
Monitoring -
Image: NG/NJ feeding tube
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As a result, a diabetes like state develops secondary to poor insulin release and increased insulin resistance.
Confirm correct positioning prior to feeding - pH testing ( Normal 1 - 5.5)
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Chest X-Ray IF uncertain
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At risk patients -
During feeding, 30-45 degree head up
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Already have poor oral intake for >5days or likely to have poor oral intake for 5 days or longer
Follow departmental feeding regimen (e.g. 30ml/hr + 4hrly aspirate. If aspirate <250ml,
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Poor absorptive capacity (Absorptive disorders, obstruction, short bowel syndrome)
safe to increase volume of feed)
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High nutrient losses (High stoma outputs/high NG aspirates)
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Increased nutritional needs due to critical illness/ Surgery
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Naso-Jejunal feeding -
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The most commonly used nutritional screening tool - MUST (Malnutrition Universal Screening tool - BAPEN)
Indications -
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In an acutely unwell patient, hyPOalbuminaemia is a marker of disease severity, not malnutrition.
Gastroparesis not responding to prokinetic agents
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Post-operative patients with upper G.I anastomosis
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Monitoring - Same as NG feeding except, aspiration is not possible with NJ feeding
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tubes
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Image: Correct NG feeding tube position - visible
Tube Enterostomy -
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guidewire (Case courtesy of Dr Ian Bickle,
Indications -
Radiopaedia.org, rID: 29342)
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Patients requiring prolonged nutritional support (>4weeks)
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Access via nasal/oral route is not feasible - Increased risk of aspiration or major Upper
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GI resection
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Can be placed using surgical, endoscopic (Percutaenous Endoscopic Gastrostomy -
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P.E.G is most common) or radiological techniques
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Monitoring -
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Can be used within 4 hours of insertion
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Regimen is the same as for NG/NJ feeding
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Flowchart: Methods of nutritional support (Adapted from chapter on nutrition, CcRISP textbook 2017)
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PARENTERAL NUTRITION
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Indications - Patients with or at risk of malnutrition in whom;
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1.Oral/enteral intake is unsafe or inadequate to meet nutritional requirements
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2.Non-functional, inaccessible or perforated gastro-intestinal tract
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Mechanical SBO, Intestinal fistulae, Short bowel syndrome (<300cm functional small bowel) or refractory I.B.D.
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Administration - All in one preparations usually given as continuous infusions (16hrs or 24hrs). See image on left.
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Monitoring - Regular dietician review
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1.Regular blood sugar measurement (B.D) - HyPERglycemia is common with TPN
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2.Daily U+Es - Risk of re-feeding syndrome (Below)
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3.Once weekly Triglyceride levels - TPN is a lipid emulsion
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4.LFTs - abnormal LFTs are seen with TPN (especially if duration >4weeks)
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Complications of Parenteral feeding -
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1. Catheter related
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Infection - Ideally catheter removal is advocated however where catheter benefit outweighs risk, dual blood and
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line cultures should be sent + microbiology advice sought early.
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Line blockage – Can be due to clotted blood or TPN in line, kinking of the line or external compression due to
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venous thrombosis. Flush with sterile 0.9% saline. Occasionally, thrombolytic agents (Alteplase) are needed.
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Central venous thrombosis – Doppler ultrasonography and managed with anticoagulants.
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2.Metabolic
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Fluid-Overload - Maintenance fluids are not usually required once a patient is fully established on TPN
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HyPERglycemia - Common occurrence with stress response and with TPN. Can use Variable rate insulin infusion.
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HyPERtriglyceridemia - Lipid emulsion in TPN poses a risk of pancreatitis.
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3.RE-FEEDING syndrome - Characterised by marked hyPOphosphatemia following rapid re-introduction of
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carbohydrate (Enterally or parenterally) in a malnourished patient. Can be FATAL.
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Pathophysiology - During malnutrition, fat and protein metabolism provides energy supply. Intra-cellular mineral
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stores become severely depleted over time. Introduction of carbohydrates results in release of Insulin and
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stimulation of glycogen, fat and protein synthesis. These require a supply of minerals (Phosphorus, calcium,
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magnesium and potassium) which become progressively depleted.
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Diagnosis - No diagnostic test. High index of suspicion
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Management - Correction of mineral and vitamin deficit. Initiation of feeding should always be under dietetic
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guidance.
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1.Central TPN - Due to HIGH osmolality, MUST be administered into a central vein to avoid thrombophlebitis.
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2.Peripheral TPN - Low osmolality TEMPORARY alternative to central TPN. Can be delivered via blue cannula BUT
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NOT other cannulas.
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